The Biology of Mental Disorders
Monday, March 30, 2009
The Biology of Mental Disorders
This would be funny if it were not so sad. There is very little biology in this book. Have a look through yourself. We think you'll find there is a lot on government policy (pork) in the document, though.

The Biology of Mental Disorders
The Biology of Mental Disorders
Sunday, March 29, 2009
Antibiotics Versus Iodine

Antibiotics are unsafe (package insert information can be found here). Some can promote cancer (fungus connection) and others, like Prozac and Accutane, can make you lose contact with reality. What's left? Iodine. Link
Iodine makes good nutritional sense. Read about it at the United Nations Standing Committee on Nutrition, here.
Saturday, March 28, 2009
Pharma Corruption: Greed Kills the Golden Goose

March 5, 2009: Globe and Mail columnist claims there is no Big Pharma conspiracy and that medicine is basically good
Nothing to see here, move along...

March 28, 2009:
Harvard shrinks issued subpoenas
Philip Dawdy of http://www.furiousseasons.com sums it up nicely:
"Controversial Harvard child psychiatrist Joseph Biederman and his colleagues Tim Wilens and Thomas Spencer were named in a federal subpoena yesterday. The trio have all pulled in millions from pharma companies while also doing federally-funded research, a potentially illegal conflict of interest which was allegedly improperly reported. They are all being investigated by their university and NIH.
The subpoena, issued by the US Attorney for Eastern Massachusetts, was served on Fletch Trammel, an attorney representing various states attorneys general and other plaintiffs in cases against pharma companies over pushing antipsychotics for use in children and adults. Apparently, the feds want whatever documents and depositions Trammel has gathered on the three."
Nothing to see, yet, but we hear the crash of distant cash registers, and expect fireworks and a feast of roast goose soon.
Thursday, March 26, 2009
Wednesday, March 25, 2009
Take Zinc for Your Thyroid

Zinc and Hormones
Zinc deficiency affects hypothalamic pituitary thyroid function. Thyrotropin releasing hormone content was decreased in the zinc deficient rat (Morley et al., 1980). Ultimately triiodothyronine (T3) and thyroxin levels were decreased. The hypothalamic axis susceptibility to Zn deficiency may explain the dynamic relationship between testosterone and Zn. Injections of testosterone or dihydro-testosterone in mice restores normal zinc content (Donovan and Thomas, 1980) while Zn deficiency decreases serum production and delays puberty (Prasad, 1966).
Elevations in hypothalamic Zn concentrations in the rat appear to correlate with the release of gonadotropin releasing hormone and gonadotropins which occurs between proestrus and estrus and after castration, although this, of course, does not establish a causal relation (Zinc and Manganese in the Schizophrenias
Merriam et al., 1979). Hypothalamic Zn ions rise with gonadotropin secretions (Merriam et al., 1979; Root et al., 1979). Hypogonadism occurs with Zn deficiency (Caggiano et al., 1969; Prasad, 1966).
Zinc deficiency inhibits essential fatty acid metabolism to prostaglandins (PG) either by blocking linoleic acid desaturation to gamma - linoleic acid or by inhibiting mobilization of dihomo - gamma linolenic acid from tissue membrane stores (Cunnane and Horrobin, 1980). Prolactin and Zn have similar actions on PGE1 formation and prolactin enhances flow of fluid from fetal to maternal compartments (Manku et al., 1975; Manku et al., 1979). Zinc deficiency can result in polyhydraminos (Manku et al., 1979). Opiates may have an effect on PGE1 synthesis which is opposite to that of Zn and there is evidence that reduced PGE1 production (possibly due to an endogenous opioid) may play a role in schizophrenia (Horrobin et al., 1978; Horrobin and Morgan, 1980). An enkephalin degrading amino peptidase from rat brain homogenates is a Zn metalloenzyme(Schnebli et al., 1979). A higher than normal proportion of arachidonate was found in the fatty acids of Zn deficient skin (Bettger et al., 1980). PGE2 and PGF2 have opposing effects on Zn transport and may act as regulators of the intestinal mucosa transport of Zn (Song and Adham, 1979).
Zinc deficiency and thyroid hormone shortage occurring in both cretinism and myxedema have similar signs, ie. retarded growth, reduced appetite and activity, impaired development of skin and hair (Hartoma et al., 1979). Zinc deficiency symptoms may be mediated by excess glucocorticoids since Zn depletion results in elevation of glucocorticoids. Elevated glucocorticoids and Zn deficiency both result in death of thymic lymphocytes (Donovan and Thomas, 1980). A deficiency of nerve growth factor may occur with Zn deficiency. One nerve growth factor is a small basic protein with three distinct types of sub-units (Vinores and Guroff, 1980). Two molecules of Zn are present in the complex and Zn participates in holding the structure together (Dunn et al., 1980; Pattison and Dunn, 1975). In the absence of Zn the subunits separate. Nerve growth factor is required for the survival and development of certain sympathetic and sensory neurons. It is equally clear that nerve growth factor affects a wide variety of other cells as well. Nerve growth factors are present on the plasma membrane and almost certainly at the synaptic ending as well (Dunn et al., 1980). Nerve growth factor action increased dendritic attachments which requires elevated levels of RNA synthesis, which is Zn dependant.
- From article on Zinc and Manganese in the Schizophrenias by Carl Pfeiffer
Link
Want to know more?
We like the About.com site hosted by Mary Shimon
here.
Listen to an mp3 about your thyroid, zinc and other minerals and vitamins from Wellness Resources here
Saturday, March 21, 2009
Chemo Brain: Cognitive Effects of Chemotherapy

"The systems of the body most affected by chemotherapy drugs include visual and semantic memory, attention and motor coordination.[6] These effects can impair a chemotherapy patient's ability to understand and make decision regarding treatment, perform in school or employment and reduce quality of life.[6]
Breast cancer survivors who were treated with chemotherapy have to work harder to perform tasks than survivors whose treatment was surgical. A year after treatment the brains of cancer survivors treated with chemotherapy had physically shrunk while those of people not treated with chemotherapy had not.[7]
Post-chemotherapy cognitive impairment comes as a surprise to many cancer survivors. Often, survivors think their lives will return to normal when the cancer is gone, only to find that the lingering effects of post-chemotherapy cognitive impairment impede their efforts. Working, connecting with loved ones, carrying out day-to-day tasks—all can be very challenging for an impaired brain. Although post-chemotherapy cognitive impairment appears to be temporary, it can be quite long-lived, with some cases lasting 10 years or more.[8]"
Link
Discussion at NPR
Friday, March 20, 2009
Charles Manson's Biochemistry

Image: Charles Manson at 74. Charles Manson may be twice a prisoner - of the California correctional system and of his own biochemistry. He has a condition known as Pyroluria which can result in odd or violent behavior, and which is easily corrected with inexpensive nutritional elements such as B6 and zinc. There seems to be a familial connection, discussed here.
Read about what is wrong with Manson's biochemistry - and that of many others - and how to correct it, in Dr. Walsh's presentation here - pdf. More on Pyroluria here.
Another page discussing a pyroluria protocol is here.
If all children were given an opportunity to be screened for nutritional type and appropriate accommodations made, crime rates would probably plummet. Let's do it!
Subscribe to:
Posts (Atom)